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Geoffrey-Burnstock

Geoffrey-Burnstock

University College Medical School UK

Dionisio-Figueiredo

Dionisio-Figueiredo

Hospital de Urgencia Otavio Lage Brazil

Ana-Frances

Ana-Frances

Valencia University Spain

Galina-Mindlin

Galina-Mindlin

Mount Sinai USA

Natalia-Verez-Cotelo

Natalia-Verez-Cotelo

San Pablo CEU University Spain

Sybille-Krauss

Sybille-Krauss

Deutsches Zentrum für Neurodegenerative Erkrankungen Germany

Peter-Silburn

Peter-Silburn

The University of Queensland Australia

Fahad-AlKherayf

Fahad-AlKherayf

University of Ottawa Canada

Alzheimers Disease - 2022

Welcome Message

On behalf of our Organizing Committee member, we welcome all distinguished guests and delegates to the 16th International Conference on Alzheimer’ and Neurological Diseases which is scheduled to take place in Prague, Czech Republic during May 04-05, 2022.

Alzheimer’s disease 2022 is going to deliver an outstanding exchange platform of ideas and authoritative views by leading scientists & researchers which covers the entire spectrum of research in Alzheimer’s disease and Neurological Disorder and share the cross-cultural experiences of various treatment procedures.

The outline of the Alzheimer’s Disease 2022 conference is, Research and Development: Alzheimer’s and Neurological Disease, with this theme it will achieve its goal under the guidance and assistance of the experienced researcher as well as young researchers.

About Conference


Alzheimer’s disease 2022 is an annual meeting of Neurologists as well as Neurology societies to discuss the future of the Alzheimer’s disease and Neurological Disorders in terms of collaboration, structures and organizational development and advances of Neurology.

The 2 days meeting is a summit to look forward for its edifying symposiums and workshops from renowned specialists of the sector and remarkable keynote sessions are coordinated by the most effective within the business.

We have also introduced awards to motivate and inspire young researcher and students to become an effective part of this immense summit, such as Best young researchers, Best Speaker, Best Keynote Session etc.

It'll likewise demonstrate to be a splendid open entryway for the agents from Universities and Institutes to collaborate with the world-class analysts and an exceptional open door for organizations sharp at extending their worldwide market reach. Interested people can affirm their interest by enrolling for the meeting alongside their partners.

Why to attend

We will endeavour to make Alzheimer’s disease 2022 a successful summit with your support and presentation on Neurological Disorder and Alzheimer’s disease. During the conference, we assure you that you will experience world class facilities and hospitality at the conference.

The Conference will feature many exciting academic programs including multiple panels, workshops, affinity group lunches, paper presentations, and scientific sessions by reputed neurologists and neurosurgeons from all over the world.

We will provide:

  • IOCM Certification
  • One on One Meeting with Eminent Speakers from 40 Countries
  • Live Streaming of the conference
  • Keynote sessions by world’s most eminent researchers at Alzheimer’s disease and Dementia research
  • Top Industries Representation
  • Thought provoking Symposia’s and Workshops
  • Preconference workshops in all major countries
  • Nominations for Best Poster Award
  • Outstanding Young Researcher Award
  • Group Registration benefits

Your attendance and participation will be greatly appreciated as we seek to strengthen the expertise in field of Neurological Disease!!

 

Sessions/Tracks

Track 01: Neurodegenerative Disease

Neurodegenerative disease is an umbrella term for a variety of conditions which primarily affect the neurons within the human brain. Neurodegenerative diseases represent a serious threat to human health.

Neurons are the elementary unit of the nervous system which incorporates the brain and spinal cord. Neurons cannot reproduce or replace themselves, so once they get damaged or die then they cannot be replaced by the body.

Neurodegenerative diseases are incurable and debilitating conditions that end in progressive degeneration and / or death of nerve cells. This causes problems with movement, mental functioning and affect an individual's ability to maneuver, speak and breathe.

Track 02: Ataxia

Ataxia means without coordination. People with this neurological disorder lose muscle control in their arms and legs. This may cause deficiency of balance, coordination, and trouble walking. Ataxia could have an affect on the fingers, hands, arms, legs, body, speech, and even eye movements.

Persistent ataxia usually happens due to the damage in the part of our brain that controls muscle coordination (cerebellum). Many conditions can cause ataxia, including alcohol consumption, certain medication, stroke, tumor, cerebral palsy, brain degeneration and multiple sclerosis.

Treatment for ataxia depends on its cause. Adaptive devices, such as walkers or canes, might help you maintain your independence. Physical therapy, occupational therapy, speech therapy and regular aerobic exercise also might help.

Track 03: Dementia

Dementia isn't a particular disease, it's a bunch of conditions characterized by impairment of a minimum of 2 brain functions, like amnesia and judgment, it happens typically once the brain get injured or the other diseases. A diagnosis of dementia requires an amendment from a person's usual mental functioning and a greater psychological failure than what is caused by traditional aging. Several diseases and harmness to the brain, such as a stroke, can give rise to dementia. But the most common cause is Alzheimer's disease, a neurodegenerative disorder.

Track 04: Alzheimer’s disease

Alzheimer’s disease (AD), likewise referred to just as Alzheimer's, is a chronic neurodegenerative illness that typically begins gradually and get serious over time. It is the reason for 60% to 70% of cases of dementia. The most widely recognized early side effect is trouble in recollecting recent event short-term memory loss. As the disease getting worse, indications can incorporate issues with language, disorientation (counting effectively getting lost), mood swings, and loss of inspiration, not overseeing self-care, and conduct issues. As an individual's condition decreases, they frequently pull back from family and society. Slowly, bodily function is lost, at last leading to death.

Track 05: Parkinson’s disease

Parkinson’s disease (PD) is the second common most neurodegenerative disease. It generally manifest as bradykinesia, rigidity, resting tremor and posture instability. It is primarily characterized by death of dopaminergic neurons within the substantia nigra, a vicinity of the mesencephalon. Notably, alpha-synuclein-ubiquitin complexes and aggregates are perceived to accumulate in Lewy bodies inside affected neurons. It is thought that deficiency in protein transport machinery and regulation might play a vital role in this disease mechanism. Impaired nerve fiber transport of alpha-synuclein may also lead to its accumulation in Lewy bodies. Membrane injury by alpha-synuclein could be an another Parkinson's disease mechanism.

Track 06: Huntington's disease

Huntington's disease (HD) is a rare chromosome dominant neurodegenerative disorder caused is caused by polyglutamine tract enlargement in the huntingtin gene, resulting in the mutant huntingtin gene (HTT). It is characterized by loss of medium spiny neurons and astrogliosis. The primary brain region to be substantially affected is the striate body, followed by degeneration of the frontal and temporal cortices. The striatum's subthalamic nuclei send control signals to the Globus Pallidus that starts and modulates motion.    

Track 07: Multiple Sclerosis

Multiple sclerosis is a chronic enervating demyelinating disease of the central nervous system, caused by an immunological attack resulting in the progressive loss of myelin sheath on neuronal axons. The resultant decrease in the speed of signal transduction leads to a loss of functionality that includes both cognitive and motor impairment depending on the location of the lesion.

The progression of multiple sclerosis occurs due to episodes of increasing inflammation, which is proposed to be due to the release of antigens such as myelin oligodendrocyte glycoprotein, myelin basic protein, and proteolipid protein, causing an autoimmune response. Multiple sclerosis presents itself as a spectrum based on the degree of inflammation; a majority of patients suffer from early relapsing and remitting episodes of neuronal deterioration following a period of recovery.

Track 08: Amyotrophic Lateral Sclerosis

Amyotrophic lateral sclerosis (ALS or Lou Gehrig's disease) is a disease in which motor neurons are particularly battered for retrogression. Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disorder that negatively impacts the upper motor neurons (UMNs) and lower motor neurons (LMNs).

It is diagnosed by skeletal muscle fragility that progresses gradually. Early diagnosis of ALS is difficult than the other neurodegenerative diseases as there are no highly effective means of determining its early onset. Currently, there is research being done relating to the diagnosis of ALS through Upper Motor Neuron tests.

Track 09: Prion Diseases

Prions are malfunctioning proteins with the ability to transmit their misfolded shape onto normal variants of the same protein. They characterize several lethal and infective neurodegenerative diseases in humans and many other animals.

Prions cause neurodegenerative disease by accumulating extra cellular within the central nervous system to form plaquette known as amyloids, which disrupt the normal tissue shape. This disruption is characterized by "holes" in the tissue with resultant spongy architecture due to the vacuole formation in the neurons.

While the incubation period for prion diseases is relatively long about 5 to 20 years, once symptoms appear the disease progresses rapidly, leading to brain damage and death.

Track 10: Batten disease

Batten disease is a rare and lethal receding neurodegenerative disorder that begins in childhood. Batten disease is the common term for a group of lysosomal storage disorders known as Neuronal Ceroid Lipofuscinoses (NCLs) – each caused by a specific gene mutation, of which there are thirteen. Since Batten disease is quite rare, its worldwide prevalence is about 1 in every 100,000 live births. It typically manifest between the ages group of 4 to 7 year. Batten disease is characterized by motor impairment, epilepsy, dementia, vision loss, and shortened lifespan. A loss of vision is the first sign of Batten disease.

Batten disease diagnosis depends on a conflation of many criteria: clinical signs and symptoms, evaluations of the eye, electroencephalograms (EEG), and brain magnetic resonance imaging (MRI) results.

Track 11: Alzheimer’s disease Diagnosis

Alzheimer's disease is generally diagnosed based upon the person's medical history, history from relatives, and behavioral observations. The presence of characteristic neurological and neuropsychological option and the absence of other conditions support the diagnosis. Advanced medical imaging with Computed Tomography (CT) or Magnetic Resonance Imaging (MRI), and the Single-Photon Emission Computed Tomography (SPECT) or Positron Emission Tomography (PET), can be accustomed to help in excluding other cerebral pathology or subtypes of dementia.

Track 12: Alzheimer’s disease Imaging

Doctor and scientists look into the brain’s grey matter while investigating about Alzheimer’s disease. A recent study, found that degenerating white matter in the brain could be an early indicator of disease. A study was published in Radiology which determines that white matter plays an important role in how the disease incurs and progresses. It can also be characterized by contraction of brain tissues due to neurons loss.

Track 13: Insulin Resistance on the development of Alzheimer’s disease

Convincing presymptomatic and clinical confirmation fortifies a pathophysiological association between Alzheimer's disease and diabetes. Reformed digestion, aggravation, and insulin resistance are key neurotic components of the two conditions. For a long time, it was considered that the cerebrum was unsympathetic to insulin, however it is presently acknowledged that this hormone has crucial neuromodulator capacities, integrating parts in learning and memory, which are enervated in Alzheimer's Disease. Despite that the sub-atomic components representing cerebrum insulin resistance in Alzheimer have remained complicated.

 

Track 14: Alzheimer’s disease and Type 2 Diabetes

Alzheimer's disease (AD) and Type 2 Diabetes Mellitus (T2DM) are extremely dominating developing related disease related with huge somberness and mortality. Few findings in human and animal models have connected T2DM to AD-type dementia. Despite epidemiological association between the T2DM and Alzheimer disease, the interpretational instruments are unclear.

T2DM is also related with cardiovascular and cerebrovascular infection (CVD), and is related with prolonged risk of infarcts and little vessel infection in the brain and various organs. Neuropathological evaluation of posthumous cerebrums demonstrated proof of cerebrovascular disease and no relationship among T2DM and β-amyloid stores or neurofibrillary tangles.

Track 15: Vascular Dementia

Vascular dementia is a common term describing problems with cerebral, planning, judgment, memory and other thought processes caused by brain injury from impaired blood flow in our brain. Vascular dementia can manifest after a stroke blocks in an artery in your brain, but strokes don't always cause vascular dementia. Whether a stroke affects our thought process and cerebral depends on the stroke's severity and location. Vascular dementia can also outcome from other circumstances that damage blood vessels and reduce circulation, depriving your brain of vital oxygen and nutrients.

Market Analysis

Alzheimer’s is a form of disease that affects the functioning of the brain resulting in decline in memory or other thinking skills. Alzheimer’s disease is the most common and largest form of dementia (in over 60% cases), which is observed among people aged 65 years and above. Accumulation of a protein called beta amyloid, in the spaces between nerve cells due to genetic mutation is a major cause of dementia. Although dementia disease is not treatable, symptomatic treatment is followed in all cases. Research is being carried out to develop treatment centered on the role of beta-amyloid in dementia.

The global dementia drugs market estimated to be valued at US$ 13,900.0 million in 2017 and is expected to witness a CAGR of 8.4% over the forecast period (2018–2026).

GlobalData estimated sales of Alzheimer Disease therapeutics in 2016 to have been around $3 billion over the seven major markets (7MM), which are the US, the five major European markets (5EU: France, Germany, Italy, Spain, and UK), and Japan. By 2026, Global Data foresees the AD market will have developed at a solid Compound Annual Growth Rate (CAGR) of 17.5%, arriving at sales of $14.8 billion over the 7MM. This is mostly credited to the developing pervasiveness of both AD and mild cognitive impairment (MCI) and the quick take-up of biologics and other novel disease therapies (DMTs).

To Collaborate Scientific Professionals around the World

Conference Date May 04-05, 2022

For Sponsors & Exhibitors

[email protected]

Speaker Opportunity

Past Conference Report

Supported By

Journal of Alzheimers Disease & Parkinsonism Journal of Neurology and Neuroscience Journal of Spine & Neurosurgery

All accepted abstracts will be published in respective Conference Series LLC LTD International Journals.

Abstracts will be provided with Digital Object Identifier by